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Catalyst #17 Pg. 87 - Take out Flashcards

Catalyst #17 Pg. 87 - Take out Flashcards. Go to the class website Open and do the Catalyst: Anatomy: Lesson 85- Catalyst #17-4/3-4/4 AND ANATOMY: LESSON 83- Drugs on the Brain PPT-4/3-4/ 4 AND Anatomy: Lesson 85- Drugs Graphic Organizer-4/3-4/ 4 AND

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Catalyst #17 Pg. 87 - Take out Flashcards

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  1. Catalyst #17 Pg. 87-Take out Flashcards Go to the class website Open and do the Catalyst: Anatomy: Lesson 85- Catalyst #17-4/3-4/4 AND ANATOMY: LESSON 83- Drugs on the Brain PPT-4/3-4/4 AND Anatomy: Lesson 85- Drugs Graphic Organizer-4/3-4/4 AND Anatomy: Lesson 85- Brain Dissection Pre Lab-4/3-4/4

  2. Fight-Or-Flight: Rest-and-Digest:

  3. (2) Postsynaptic neuron (1) Presynaptic neuron (3) neurotransmitter

  4. Announcements • Brain Dissection: 4/7-4/8 • Pre lab needs to be completed in order to be part of the dissection • You will be assigned to groups of 4 • Dissection= 1 test grade (you cannot make this up) • Nervous System Test: 4/11-4/12 • 1 test grade • Notebook Check Date Change AND Grade Change: • Wednesday 4/10 • Count as 2 grades for March AND April • TOTAL: 4 grades

  5. Let’s Explore (20-30 min)-Pg.90 • https://faculty.washington.edu/chudler/introb.html#dr • Directions: • Click on 5 drugs • Read the entire page • Fill out your graphic organizer

  6. Explain the different categories of drugs and their mechanisms of action.

  7. Get Ready for Notes Pg. 88-89Title: Drugs on the Brain

  8. What is a drug? • Any substance that produces a change in the structure or function of your body • A psychoactive drug produces a change in the structure of your function of your brain

  9. Categories of psychoactive drugs

  10. Sympathetic Nervous System

  11. 1. stimulant • Speeds up body processes by acting on the sympathetic nervous system (sympathetomimetics);“upper” • Based on the above, what symptoms would you predict?

  12. 1. stimulant • Speeds up body processes by acting on the sympathetic nervous system (sympathetomimetics);“upper” • Symptoms • Abnormal cheerfulness or talkativeness • Frequent urination • Loss of appetite • Difficulty sleeping • Increased heart rate & breathing • Anxiety

  13. 2. depressants

  14. 2. depressant • Slows down body processes by acting on parasympathetic nervous system (“downers”) • Symptoms • slows down breathing and heart rate • slows down reflexes • disrupts coordination • Impairs judgment • lack of concentration

  15. Stop & Think Compare and contrast stimulants versus depressants

  16. 3. opiate • Endorphins make you happy • Opiates are agonists for endorphins, and are used a painkillers

  17. 3. opiate • Symptoms • Produce a sense of euphoria and well-being, blocking out pain and problems • Drowsiness • Lack of attention • Loss of appetite • Extremely addictive • Your body: “You don’t need me to make endorphins anymore, right?”

  18. 4. hallucinogens • “psychedelics” • Changes your perceptions of things: sensory hallucinations, loss of identify, vivid fantasies • Persistent: reverse tolerance • Disassociatives: A type of hallucinogen that blocks signals to the conscious mind from the rest of the brain

  19. 5. uncategorized • Some drugs are complex and have characteristics of multiple categories • Examples of these drugs include marijuana and ecstasy

  20. Agonist vs. Antagonist: Return of the Synapse neurotransmitter receptor synaptic vesicle neurotransmitter synaptic cleft presynaptic neuron (axon terminal) postsynaptic neuron (dendrite)

  21. Action Potential Arrives neurotransmitter receptor synaptic vesicle neurotransmitter synaptic cleft presynaptic neuron (axon terminal) postsynaptic neuron (dendrite)

  22. Synaptic Vesicle merges membrane neurotransmitter receptor synaptic vesicle synaptic cleft presynaptic neuron (axon terminal) postsynaptic neuron (dendrite)

  23. Neurotransmitters released into synaptic cleft neurotransmitter receptor synaptic vesicle synaptic cleft presynaptic neuron (axon terminal) postsynaptic neuron (dendrite)

  24. Neurotransmitters released into synaptic cleft (diffusion) neurotransmitter receptor synaptic vesicle synaptic cleft presynaptic neuron (axon terminal) postsynaptic neuron (dendrite)

  25. Neurotransmitters bind to receptors on the post synaptic neuron neurotransmitter receptor synaptic vesicle synaptic cleft presynaptic neuron (axon terminal) postsynaptic neuron (dendrite)

  26. This binding causes an excitation or an inhibition neurotransmitter receptor synaptic vesicle Excitation more action potentials synaptic cleft presynaptic neuron (axon terminal) postsynaptic neuron (dendrite)

  27. Neurotransmitter reuptake (“recycling”) neurotransmitter receptor synaptic vesicle neurotransmitter synaptic cleft presynaptic neuron (axon terminal) postsynaptic neuron (dendrite)

  28. This binding causes an excitation or an inhibition glutamate neurotransmitter receptor excitation more action potentials GABA inhibition fewer action potentials postsynaptic neuron (dendrite)

  29. An agonist is a “pretender” neurotransmitter receptor synaptic vesicle neurotransmitter synaptic cleft presynaptic neuron (axon terminal) postsynaptic neuron (dendrite)

  30. An antagonist blocks the receptor neurotransmitter receptor synaptic vesicle neurotransmitter synaptic cleft presynaptic neuron (axon terminal) postsynaptic neuron (dendrite)

  31. A drug pretends to be serotonin, binding to serotonin’s postsynaptic receptors and causing action potentials.

  32. A drug pretends to be serotonin, binding to serotonin’s postsynaptic receptors and causing action potentials.

  33. Alcohol blocks glutamate receptors so that signaling cannot happen.

  34. Alcohol blocks glutamate receptors so that signaling cannot happen.

  35. Cocaine/Crack The drug block the reuptake channels for dopamine. When dopamine is released, it is unable to return back to the pre-synaptic neuron. This causes more neurotransmitter to be released into the synaptic cleft, more sodium channels are open, and more action potentials are fired.

  36. LSD The drug most resembles the neurotransmitter serotonin, so it is able to unlock the postsynaptic receptor. This causes more action potentials (electric signaling) in the visual cortex of the brain.

  37. Meth This drug enters the pre-synaptic neuron through the reuptake channels and takes the place of dopamine in the synaptic vesicles. The dopamine is then bullied out and pushed into the synaptic cleft causing an overflow of the neurotransmitter.

  38. Alcohol • This drug increases the effect of GABA, an inhibitory neurotransmitter. It slows down action potentials. • The other way is that it blocks post-synaptic receptors for glutamate so that the glutamate cannot bind to postsynaptic receptors and release a signal.

  39. Marijuana • Cannabinoid receptors exist in the hippocampus, cerebellum • Cannabinoid receptors are activated by a neurotransmitter called anandamide. • THC mimics the actions of anandamide, meaning that THC binds with cannabinoid receptors and activates neurons, • Blocks acetylcholine from being released.

  40. Brain Dissection Pre Lab- Separate sheet of paper that you will turn in on Mon/Tues • Needs to be complete in order to do lab on Mon/Tues • REMEMBER: • You cannot make up the brain lab • Unexcused Absent= NP

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