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Ophthalmic Emergencies

Ophthalmic Emergencies. How to avoid missing a serious and treatable condition. Hong Woon SJUH, Leeds. Ophthalmic Emergencies. Ophthalmic emergencies with obvious clinical signs Ophthalmic emergencies without obvious clinical signs. Alkali burns.

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Ophthalmic Emergencies

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  1. Ophthalmic Emergencies How to avoid missing a serious and treatable condition • Hong Woon SJUH, Leeds

  2. Ophthalmic Emergencies • Ophthalmic emergencies with obvious clinical signs • Ophthalmic emergencies without obvious clinical signs

  3. Alkali burns • Fertilizers and cleaning products/ drain and oven cleaners/ cement • Must treat immediately with irrigation before referring on. • Irrigate with copious amounts of water/ buffer solution until pH normal • Untreated can become blind due to stromal damage and ischaemia

  4. Beware infection in CL wearer of if recent operation • Painful red eye: - may indicate infective keratitis/ endophthalmitis • Urgent treatment required

  5. Dendritic ulcers • Self limiting • Problem occurs if treated with topical steroids

  6. Ophthalmic emergencies without obvious clinical signs Choices: • Make diagnosis from examination • Become competent at slit lamp and fundus examination • Make diagnosis from symptoms • Learn to take a good history

  7. What to ask for when taking a history of visual loss? • Character of visual loss may help identify location of disease: • Extent of visual loss: sharpness / darkness / field loss • Blurred vision: Out of focus / Distorted vision • Floater • Glare / Halos • Timing: onset / progressive / transient • Associated factors: pain / headache

  8. Extent of visual loss Very important clue about severity of problem Ask about vision of each eye Vision of less than CF is indicative of serious disease But remember serious disease can also be present if vision is good

  9. Quantify extent of visual loss with vision test • Sit patient at correct distance from chart • Test each eye individually • Ask patients to use distance glasses • Use pinhole if no glasses • If unable to see any letters • ? CF, ?HM, ?PL • Compare with previous vision

  10. Blurred vision • Out of focus / not sharp • No distortion • Refractive problem • Cornea • Lens • Shape of eye • Macular problem

  11. Distortion of vision(Metamorphopsia) • Things look wavy, jumbled up. Lines not straight. Kink in lines • Condition affecting retina • Wet macular degeneration • Macular hole • Macular pucker • Retinal detachment

  12. Field defect • Curtain, bit missing, bump into things • Always a sign of serious problem • If homonymous: -same visual field both eyes • Defect of visual pathways • Not hononymous • Retinal • Optic nerve

  13. Shadow in the eye • Retinal detachment • Glaucoma • Vitreous haemorrhage • Vascular lesion • Optic nerve disorder • Visual pathway problem

  14. Floater • Due to opacity in vitreous • Fly/spider, blob, haze • Movement

  15. Floaters PVD May notice Weiss’ ring Syneresis Vitreous haemorrhage Rain drops Black streaks Posterior uveitis

  16. Flashes PVD Moores lightening streak Peripheral scintillation with eye movement Migraine Scintillating scotoma Fortification spectrum Mass lesion Retinal ischaemia

  17. Glare • Difficulty seeing in bright light • Low sun • Driving at night • Fluorescent light in supermarkets • Corneal or lens problem • Often due to cataract • Vision may be good otherwise

  18. “Halos” in acute glaucoma Not strictly a halo – diffraction of light by ice crystals Coloured rings around lights due to refraction by water droplets Not to be confused with glare due to scatter by corneal or lens opacities

  19. Reduced colour sensitivity • Things look darker / less colour • Optic nerve disease • Optic neuritis • Compressive optic nerve disease • Condition affecting retina • Wet macular degeneration • Central serous retinopathy

  20. Timing of visual loss • Recent or chronic • Static or progressive • Transient or persistent • Very important in determining urgency of situation

  21. Conditions causing sudden visual loss • Vascular occlusion • Central retinal artery occlusion • Central retinal vein occclusion • Anterior ischaemic optic neuropathy • Vitreous haemorrhage • Acute glaucoma Conditions causing rapid progressive visual loss • Retinal detachment • Wet macular degeneration

  22. Causes of transient visual loss • Amaurosis fugax • Subacute angle closure glaucoma • Visual obscuration due to papilloedema • Giant cell arteritis • Floaters • Ocular surface problem

  23. Symptoms suggestive of urgent condition • Marked visual loss • Field loss • Floaters/Photopsia • Distortion of vision • Transient marked loss of vision • Recent change • Rapidly progressive change

  24. Ophthalmic emergencies without obvious clinical signs • Subacute angle closure glaucoma • CRAO • AION • Wet macular degeneration • PVD with retinal tear/detachment • Retinal detachment • Trauma • CRVO • Neurological conditions presenting with eye signs/symptoms

  25. Subacute angle closure glaucoma • Acute angle closure glaucoma – ophthalmic emergency with obvious clinical signs • Red eye • Painful eye • Severe visual loss in affected eye • Often premonitory episodes of subacute angle closure glaucoma

  26. Subacute angle closure glaucoma • Angle closure develops in evenings when pupil dilates • Pressure increases to give pain and corneal oedema • Patient sees blurred vision and haloes around lights • Patient goes to bed • Sleep induced miosis aborts attack of glaucoma • Normal eye next day • Should be referred urgently before established AACG develops

  27. Treatment of to prevent angle closure glaucoma • YAG PI • Laser treatment • PI prevents build up of aqueous behind pupil

  28. Central retinal artery occlusion • Sudden severe visual loss • Visual loss may not be total due to cilioretinal artery • Usually embolic, rarely due to GCA • Attempt to reperfuse artery if within 24 hours of onset • Should be seen urgently to exclude GCA and identify cause • No recovery of vision • Often premonitory symptoms

  29. Amaurosis fugax • Transient visual loss due to passage of emboli through central retinal artery • Recovery to normal vision unless permanent central retinal artery occlusion develops • Often no emboli visible on clinical examination • Total or altitudinal visual loss in one eye for a few minutes • Visual recovery often described like a curtain lifting • Aspirin very effective in preventing CRAO

  30. Cause of CRAO is usually carotid disease • Risk factors: • Hypertension, Smoking, Diabetes, High Cholesterol • Risk of further emboli to eye or brain • Reduce risk factors • Antiplatelet agents • Carotid endarectomy if greater than 70% stenosis

  31. Anterior ischaemic optic neuropathy • Infarction of optic nerve head - pale swollen disc with visual loss. • Sudden severe visual loss if entire disc affected, altitudinal defect if only one short posterior ciliary artery occluded

  32. Anterior ischaemic optic neuropathy • No treatment for affected eye • 75% due to atherosclerosis in middle aged smokers • 25% due to Giant cell arteritis - headache, tenderness of temporal arteries, malaise, raised ESR . • AION due to GCA is emergency - high risk of developing AION in fellow eye

  33. Wet macular degeneration1. Serous elevation of retina or PED2. Blurred vision and distorted vision3. Usually progressive over weeks4. Difficult to see with ophthalmoscope5. Initial vision may be very good

  34. Wet ARMD • Highly likely to progress to severe visual loss if untreated • Recovery of vision best if treated before chronic changes have developed • Treatment not recommended for late cases

  35. Treatment of CNM with intravitreal injection of antiVEGF • VEGF (vascular endothelial growth factor) stimuates growth of choroidal neovascular membrane • antiVEGF binds to VEGF and prevents it acting on CNM • Visual loss can be reversed if caught early

  36. Diagnosis of posterior vitreous detachment • Flashes and floaters • Weiss’ ring • Age, myopia, cataract operation • Examination • Weiss’s ring • Posterior hyaloid membrane

  37. Symptomatic Posterior Vitreous Detachment • Risk of developing retinal tear (8-22%) • Risk of developing RD: 3 – 7% in symptomatic PVD • If RD develops, it usually occurs within 6 weeks

  38. Symptomatic Posterior Vitreous Detachment Indications for urgent referral • Recent history (? Within 6weeks) • Symptoms suggestive of vitreous haemorrhage • Symptom of retinal detachment Reason for urgent referral • To pick up retinal tear before retinal detachment • Early treatment of retinal detachment

  39. Penetrating ocular injury • Suspect if history of high velocity fragment – ie metal on metal fragment • May be minimal external eye signs.

  40. Symptoms suggestive of urgent condition • Marked visual loss • Field loss • Floaters/Photopsia • Distortion of vision • Transient marked loss of vision • Recent change • Rapidly progressive change

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