1 / 37

Trichotillomania: An Overview

Trichotillomania: An Overview. David Dia, PhD, LCSW, CCBT University of Tennessee. Disclosures. No financial disclosures or conflicts of interest to report

howe
Télécharger la présentation

Trichotillomania: An Overview

An Image/Link below is provided (as is) to download presentation Download Policy: Content on the Website is provided to you AS IS for your information and personal use and may not be sold / licensed / shared on other websites without getting consent from its author. Content is provided to you AS IS for your information and personal use only. Download presentation by click this link. While downloading, if for some reason you are not able to download a presentation, the publisher may have deleted the file from their server. During download, if you can't get a presentation, the file might be deleted by the publisher.

E N D

Presentation Transcript


  1. Trichotillomania: An Overview David Dia, PhD, LCSW, CCBT University of Tennessee

  2. Disclosures • No financial disclosures or conflicts of interest to report • Information is presented as educational. It is not intended to diagnosis, treat, or be a substitute for expert medical or mental health care.

  3. Overview • General information • Medication • Psychosocial Treatments

  4. What is in a name? • Trich = hair • Tillo = pulling • Mania = impulse

  5. Diagnosis – DSM IV-TR • Impulse Control Disorder • May be related to Obsessive compulsive disorder (anxiety) or tic disorder • Pathological Gambling

  6. Diagnosis – DSM IV-TR • Criteria • Recurrent pulling out of one’s hair with noticeable hair loss • Sense of tension before pulling or when attempting to resist • Pleasure, gratification, or relief when pulling out hair • Causes clinically significant impairment

  7. Other terms to know • Trichophagia – eating the hairs • Bezoars – hair balls • Alopecia – hair loss

  8. General Facts • Estimated 2.5 million people (.6% lifetime) • Average age of onset = 13 years old • 3.4% Females vs. 1.5% of Males hair loss • Tends to have a waxing and waning course

  9. General Facts • Can be triggered and exacerbated by anxiety • Two types, not mutually exclusive • Focused vs. automatic

  10. General Facts • Only 15% of adults experience significant improvement with community treatment! • Most (55%) believed their clinician • Did not have significant knowledge of the disorder • Did not have knowledge of evidenced based treatment

  11. Adult Sample Mood Anxiety Substance abuse Personality D/O Pediatric Sample Anxiety Depression Disruptive Behavioral Tics Comorbidity

  12. Consequences • Lower life satisfaction • Higher levels of stress • Lower self-esteem

  13. Suspected Causes • Genetic Component • 5HT2A, hoxB8, and SLITRT1 • Neurotransmitters • Dopamine • Monoamine system (MAOI) • Gultamate (precursor to GABA) • Neuroadrenaline system • Serotonin?

  14. Suspected Causes • Inferior frontal cortex – cognition • Amygdala-hippocampal formation – affect regulation • Putamen – habit learning • VTA and Nucleus accumbens • Mediates reward process

  15. Compulsive Skin Picking • General Information • 2 to 4% of the population • More common in females • Bimodal onset -- • Late childhood to early adolescents • 30 to 45 years old • Causes • Genetic (hoxb8)

  16. Overview of Treatment • No randomized control studies with pediatric • Behavioral treatments with adults demonstrate efficacy • Uncontrolled studies with pediatric show similar results as adults • SSRIs (double blind, placebo controlled) have no efficacy

  17. Overview of Treatment • Supportive or psychodynamic therapy no or minimal affect

  18. Treatment -- Medications • Mancini et al. (2009) -- pediatric • Retrospective chart review (N = 11) • 10 tried on SRI and 1 on antipsychotic • 2 on SRI and 9 on antipsychotic • 2 remitted • Results favored antipsychotics • Risperidone (Risperdol) • Quetiapine (Seroquel)

  19. Medications - Adults • SSRI vs. control condition • 3 studies • Tricyclic vs. control • Clomipramine (Anafranil) 2 studies • HRT vs. SSRI • HRT vs. Clomipramine • Clomipramine vs. SSRI (SSRIs – fluvoxamine, fluoxetine, , sertraline, citalopram)

  20. Medications – Experimental -- Adults • Opioid antagonist = Naltrexone and Nalmefene • Mood Stabilizers = lithium and Valproic acid (Depakote) • Dopamine reuptakers inhibitors = Focalin, Ritalin, Wellburtrin) • Norepinephrine inhibitor – clomipramine • Glutamatergic – N-acetylcysteine

  21. Medical • Neurosurgery, Transcranial Magnetic Stimulation (TMS), ECT • No evidence

  22. Psychosocial Treatments • HRT/CBT vs. control • 5 studies pediatric • 77% to 61 % “clinically significant changes” • For example, 16 to 5 of the MBHHPS • 5 controlled studies with adults • 91% to 61% reduction • Hypnotherapy • Two uncontrolled, small studies with adults

  23. Treatment Guidelines Pediatric • Psychoeducation • 0 to 7 years • Response prevention implemented with parents • Older than 7 years • Habit reversal therapy

  24. Treatment Guidelines Pediatric • “If there continues to be significant impairment from trich despite prolonged behavioral treatment with experienced clinician consider” • N-acetylcysteine • Initial dose 600mg, titrated to a max does of 1200mg BID • Clomipramine (Medications, including OTR, needs to be dispensed/recommended by physician)

  25. Treatment - CSP • One Double Blind Study • Fluoxetine – improvement in 2 or 3 outcome measures • Open label • Fluvoxamine, Fluoxetine, Lamotrigine, Escitalopram, N-acetylcysteine

  26. Treatment - CSP • 3 Psychosocial studies • HRT with 3 month F/U • HRT + ACT • Internet based treatment – 62% “responders” • 115 participants • 15% completed all three phases

  27. Comprehensive ModelMansueto et al. (1999) • Phase I • Assessment and functional analysis • Phase 2 • Identify and target modalities • Phase 3 • Identify and implement strategies • Phase 4 • Evaluation and modification

  28. Phase I • Two types of antecedents to pulling • Cues that trigger the urge to pull • Discriminative stimuli that facilitates pulling • Actually pulling • Consequences of pulling • Maintains pulling • Terminate pulling

  29. Phase I • Cues • External – settings and implements associated • Internal – affective states, visual or tactile sensations, cognitive cues • Discrimitive stimuli (set the stage) • External – environment free of potential observers, presences of pull instruments • Internal – urge, posture cues, cognitive

  30. Phase I • Preparation • Specific Hair selected • Disposition of hair • Discarded • Retrain • Inspect • Bite/swallow • Wrapping hair / tickle

  31. Intervention Phase I • Self-monitoring

  32. Phase 2 • Cognitive modality • Cognitive restructuring, guided self dialogue • Affective modality • Relaxation exercises, exposure, positive imagery, stress management • Motoric modality • Finger tip bandages, gloves, bracelets, eye glasses, scarf's, etc. • Silly putty, worry beads, soft brush

  33. Phase 2 • Sensory modality • Numbing cream, brushing hair, washing hair vigorously, shampoo • Gummy bears, sunflower seeds, dental floss, koosh balls, frayed blankets • Dying hair, cutting finger nails • Environmental • Removing tweezers, covering mirrors • Behavioral plans, stimulus control

  34. Phase 2 • Habit reversal • Self-monitoring • Awareness training • Hair pulling and high risk situations • Stimulus control • Decrease opportunities or interfere • Competing response intervention

  35. Phase 3 • Identify and choose treatment strategies • Client to use strategy for at least one week • Primary issue – getting the client to use strategy consistently

  36. Phase 4 • Evaluation and Modification

  37. Questions

More Related