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Temporary and Permanent Cardiac Pacing. IntroductionTemporary pacing : Indications, TechniquePermananent Pacing : Pacemaker Nomenclature Indications Selection of Pacing Mode Pacing for Hemodynamic Improvement Pacemaker Implantation, Complications Pacemaker Troubleshooting .
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1. Temporary and Permanent Cardiac Pacing
3. Temporary Cardiac Pacing Transvenous
Transcutaneous
Epicardial
Transesophageal
4. Indications for Temporary Pacing Acute myocardial infarction with:
CHB, Mobitz type 2 AV block, medically refractory symptomatic bradycardia, alternating BBB, new bifascicular block, new BBB with anterior MI
In absence of acute MI : SSS, CHB, Mobitz type 2 AV block
Treatment of tachyarrhythmias : VT
5. Temporary Transvenous Pacing Unipolar Electrograms
6. Paced QRS Morphology
7. Permanent Pacing
8. The Pacemaker System Patient
9. Pacemaker Implantation Epicardial
Transvenous :
Generator implanted anterior to pectoral muscle
Atrial/Ventricular leads via subclavian or cephalic vein
Sensing and pacing threshold
Chest X-ray for pneumothorax, lead position
10. Codes Describing Pacemaker Modes
11. DDD
12. Indications for Pacing for AV Block
13. Indications for Pacing for Sinus Node Dysfunction
14. Pacing for Hemodynamic Improvement Hypertrophic Obstructive Cardiomyopathy
Cardiac Resynchronization Therapy
Pacing to Prevent AF
Pacing in Long QT Syndrome
Neurocardiogenic Syncope
15. 15 Mode Selection Considerations
Status of Atrial Rhythm - Intrinsic vs. Paced - Presence of Atrial Tachyarrhythmias:
Acute/Chronic
Status of AV Conduction
Normal -Slowed-Blocked
Presence of Chronotropic Incompetence
16. Choice of Pacing Mode
17. Rate Responsive Pacing Goal: To provide an increased heart rate for the chronotropic incompetent patient
The Pacemaker:
Allows programming of a minimum rate and a maximum rate
Is allowed to pace (in response to sensor input) at any rate in-between this min and max rate
18. Today’s Sensors Vibration
non-physiologic
Acceleration
non-physiologic
Minute Ventilation
physiologic
Temperature
physiologic
19. Pacemaker Implantation
21. Sensing
22. Pacemaker Follow-up GOAL OF FOLLOW-UP
Verify appropriate pacemaker operation
Optimize pacemaker functions
Document findings, changes and final settings in order to provide appropriate patient management
23. Dual Chamber Pacemaker
24. Pacemaker – Magnet Application
25. Pacemaker – Mode Switch ( 1 of 3 )
26. Mode Switching in a Dual Chamber Pacemaker ( 2 of 3 )
27. Mode Switching in a Dual Chamber Pacemaker ( 3 of 3 )
28. Acute Complications of Pacemaker Implantation Venous access
Pneumothorax, hemothorax
Air embolism
Perforation of central vein
Inadvertent arterial entry
Lead placement
Brady – tachyarrhythmia
Perforation of heart, vein
Damage to heart valve
Generator
Pocket hematoma
Improper or inadequate connection of lead
29. Delayed Complications of Pacemaker Therapy Lead-related
Thrombosis/embolization
SVC obstruction
Lead dislodgement
Infection
Lead failure
Perforation, pericarditis
Generator-related
Pain
Erosion, infection
Migration
Damage from radiation, electric shock
Patient-related
Twiddler syndrome
30. Pacemaker Troubleshooting Failure to capture – high threshold,lead dislodgement, conductor coil fracture
Failure to pace ( failure to output ) – oversensing, circuit interruption, battery depletion
Failure to sense – undersensing, oversensing
31. Intermittent Loss of Ventricular Capture
32. Myopotential Sensing
34. “Pacemaker Syndrome” Fatigue, dizziness, hypotension
Caused by pacing the ventricle asynchronously, resulting in AV dissociation or VA conduction
Mechanism: atrial contraction against a closed AV valve and release of atrial natriuretic peptide
Worsened by increasing the ventricular pacing rate, relieved by lowering the pacing rate or upgrading to dual chamber system
Therapy with fludrocortisone/volume expansion NOT helpful
35. Sources of Electromagnetic Interference Medical
MRI
Lithotripsy
Electrocautery/cryosurgery
External defibrillators
Therapeutic radiation Nonmedical
Arc welding equipment
Automobile engines
Radar Transmitters
36. Expanded Indications for Pacing
Cardiac resynchronization therapy
Hypertrophic cardiomyopathy
Neurocardiogenic syncope
Long QT syndrome
Prevention of atrial fibrillation
37. Normal Conduction Is Important Normal conduction allows for prompt and synchronous activation of the atria and ventricles
Results in a brief P wave, PR interval and a narrow QRS
38. Heart FailureDelayed Ventricular Activation
39. Heart FailureBifocal Ventricular Pacing
40. Bi-Ventricular Pacing
43. Baseline ECG
44. Bi-V Pace