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Mengenali ACS: Cegah dan tangani dg cepat dan benar

Mengenali ACS: Cegah dan tangani dg cepat dan benar. Mohammad Saifur Rohman , dr.SpJP , PhD.FICA. Mengapa Penting?. Jantung adalah organ yang sangat vital Menjamin pasokan kebutuhan seluruh organ tubuh Jantung memompa lebih dari 1 juta gallon pertahun Berhenti ----- Death

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Mengenali ACS: Cegah dan tangani dg cepat dan benar

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  1. Mengenali ACS: Cegah dan tangani dg cepat dan benar Mohammad SaifurRohman, dr.SpJP, PhD.FICA

  2. Mengapa Penting? • Jantung adalah organ yang sangat vital • Menjamin pasokan kebutuhan seluruh organ tubuh • Jantung memompa lebih dari 1 juta gallon pertahun • Berhenti ----- Death • Pertanda awal  Kenali ! • Terlambat Irreversible (?)

  3. Kegawatdarutan kardiak • Cardiac arrest • Acute coronary syndrome • Cardiac dysrhythmias • Acute pulmonary oedema • Trauma – both blunt & penetrating • Cardiogenic shock • Aortic dissection • Valve insufficiency Bgm Keluhan & Tanda ?

  4. Mengapa Harus Cepat? • Serangan Jantung • Proses adaptasi terhadap perubahan yang sangat cepat • Kompensasi • Dekompensasi Dapat di kenali dan diantisipasi serta dicegah

  5. Diperbaiki? Tidak!, Dicegah? Ya !

  6. Prevalensi SKA • Di Amerika : kejadianInfarkmiokardAkut (IMA) lebih 1 Juta/tahun • 200,000 – 300,000 pasien IMA meninggalsebelumsampai RS • Total : WarganegaraAmerikamengalami IMA setiap 29 detikdanmeninggalsetiapmenit. • Indonesia ? • Tahun 2008: PJN Harapan Kita 7 pasien SKA , 50-60% IMA • !0% IMA < 40 thn Topol EJ. CV med 2009 Data PJN HK 2008

  7. Apapenyebab KEMATIAN? keterlambatanpasiendatangke RS Penanganandi RS SKA DI RSSA MALANG AMI prevalence in Saiful Anwar General Hospital 59 patients death cases among 356 patients The second deadliest diseases The mortality rate was 16,6% in 2010

  8. Seeking care behaviour pattern Patient Saiful Anwar Hospital General Practitioner role Self Medication PHC

  9. Aterosklerosis  SKA

  10. Atherogenesis

  11. Progression of Plaque toward rupture

  12. Progression of Plaque toward rupture

  13. Plaque Rupture

  14. Platelet aggregation in ruptured plaque Platelets aggregate at the site of rupture / erosion Lipid core Adventitia Weissberg, 1999

  15. Thrombus formation Thrombus forms and extends into the lumen Thrombus Lipid core Adventitia Weissberg, 1999

  16. Plaque Rupture Toward Occlusion

  17. Spektrum SKA • Unstable Angina Pectoris : (EKG normal, Trop T/I (-)) • Acute Non ST-Elevation Myocardial Infarction (NSTEMI) : (EKG normal/ST depresi/T inversi dan Trop T/I (+)) • Acute ST-Elevation Myocardial Infarction (STEMI) : EKG ST elevasi dan Trop T/I(+)

  18. Bagaimana Diagnosa SKA ?

  19. Membedakan Nyeri dada: SKA? • 1. Cardiac or non cardiac • 2. Cardiac : Ischemic non Ischemic • 3. Ischemic : Coronary non Coronary • 4. Angina pektorisstabilatau SKA

  20. Nyeri (tidak enak) dada ….. ? • Sifat :Berat/ tertindih (pressure, tightness, or heaviness, strangling, constricting, or compression),Panas (burning) ; Masukangin, Sesak,”maag” • Lokasi: Di dada kiri/tengahtidakbisaditunjuk • Penjalaran : kebahu/lengan, leher, dagu, • belakang,epigastrium • Lama : 5-30’ • Pencetus :aktifitas/stres/dingin • Berkurang: Nitrat/Istirahat • Tidakkhas: Pingsan/kejang/tidaksadar/berdebar ESC guidelines for SAP 2006 ESC AMI ST elevation guidelines 2008

  21. Angina Pectoris • A syndrome resulting from myocardial ischemia • Demand and supply imbalance • Careful history taking; mode of onset, location, quality of pain, duration, precipitating factors, pattern of disappearance, risk factor, etc

  22. Angina Pectoris • A syndrome resulting from myocardial ischemia • Demand and supply imbalance • Careful history taking; mode of onset, location, quality of pain, duration, precipitating factors, pattern of disappearance, risk factor, etc

  23. Hati-hati : Angina Equivalent • Indigestion, belching, dyspnea • DM, wanita, manula (post operative) • Didapatkan 5% dari ACS • 2% dipulangkan ternyata ACS Braunwalds Heart Disease 8th Ed 2008

  24. DD Chest pain Ischemic • Stenosis Aorta • Regurgitasi Aorta • Hypertrophic Cardiomyopathy • Angina pada Hypertensi • Hipertensi pulmonal berat 11th ed Hurst’s the heart 2005

  25. DD Chest Pain Non Ischemic • Diseksi Aorta • Pericarditis • Mitral valve prolaps 11th ed Hurst’s the heart 2005

  26. DD Chest Pain Gastro intestinal • Esophageal spasm/reflux/rupture • Peptic Ulcer Neuromusculoskeletal • Costochondritis • Herpes zoster • Chest wall pain dan tenderness etc 11th ed Hurst’s the heart 2005

  27. DD chest pain Pulmonary • Pulmonary emboli • Pneumothorax • Penumonia with pleural involvement Pleurisy Psychogenic • Axiety/depression/cardiac psychosis etc 11th ed Hurst’s the heart 2005

  28. Non Angina Pain • Hanya terasa pada sebagian kecil dada kiri/kanan (bisa di tunjuk) • Berkahir berjam jam sampai berhari hari. • Biasanya tidak berkurang dengan nitrogliserin • Mungkin dicetuskan oleh debaran. ESC guidelines for SAP 2006 ESC AMI ST elevation guidelines 2008

  29. Nyeri dada khas SKA • Angina awitan baru derajat 3 menurut klasifikasi kanada kardiovaskuler group ( nyeri dada timbul pada aktifitas ringan sehari-hari) • Angina saat istirahat > 20 menit • Perburukan derajat angina menjadi derajat 3 dalam beberapa hari – 1 bln terakhir (Crescendo angina) • Atypical

  30. PemeriksaanFisik • Sadar-Koma • TD: Hypertensi-Normal-Hypoptensi • HR: Regular-irregular/ Bradycardia-Tachycardia pulseless • RR: Tachypnea-apnea • Cor: Regular-iregular, murmur, gallop • Pulmo: Normal-Rales- wheezing • Ext: dingin/hangat, edema+/-, etc.

  31. EKG • Secepatmungkin – 10’ setelahpasientiba • Diulangapabilameragukanadanyakenaikansegmen ST (ST televasi) • Bandingkandenga EKG sebelumnya • Pasang monitor EKG

  32. EKG : Gambaran aktifitas listrik jantung

  33. EKG pada SKA EKG dapat menentukan adanya: • Old/Recent/Acute infarction • Pericarditis • Arrhythmias • Pembesaran jantung

  34. Gambaran EKG padaIskemik/IMA

  35. UAP/Acute NSTEMI

  36. Acute NSTEMI

  37. Acute STEMI- Evolution

  38. Acute STEMI-Q wave

  39. Occluded artery

  40. Anterior STEMI ECG demonstrates large anterior infarction

  41. Inferior STEMI Proximal large RCA occlusion ST elevation in leads II, III, aVF, V5, and V6 with precordial ST depression

  42. Inferior STEMI Small inferior distal RCA occlusion ECG changes in leads II, III, and aVF

  43. EKG • Gangguan Irama • Infark baru atau lama • Perikarditis • Pembesaran jantung dll

  44. 1st degree 2nd degree Type 1

  45. 2nd degree Type II 3rd degree

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