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بسم الله الحمن الرحيم

بسم الله الحمن الرحيم. (قل ان صلاتي و نسكي و محياي ومماتي لله رب العالمين لا شريك له وبدلك امرت وأنا اول المسلمين). قال رب اشرح لى صدرى ويسر لى أمرى واحلل عقدة من لسانى يفقهوا قولى. طه 25 - 28. SINUSITIS. Dr. Abdussalam M jahan ENT depart, Misurata university, faculty of medicine.

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بسم الله الحمن الرحيم

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  1. بسم الله الحمن الرحيم (قل ان صلاتي و نسكي و محياي ومماتي لله رب العالمين لا شريك له وبدلك امرت وأنا اول المسلمين) قال رب اشرح لى صدرى ويسر لى أمرى واحلل عقدة من لسانى يفقهوا قولى طه25-28

  2. SINUSITIS Dr. Abdussalam M jahan ENT depart, Misurata university, faculty of medicine

  3. Classification: • Rhinosinusitis is classified according to: • clinical presentation : • (acute, subacute, chronic), • (b)anatomic site of involvement : • (ethmoidal, maxillary, frontal, sphenoidal & pan sinusitis),

  4. Acute Rhinosinusitis: • Having a duration of less than or equal to 4 weeks. • Subacute Rhinosinusitis: • Having a duration of 4 to 12 weeks. • Chronic Rhinosinusitis: • Lasting longer than 12 weeks.

  5. Acute Rhinosinusitis: • Etiology: • presence of bacteria in a sinus cavity with an obstructed ostium. • failure of the mucociliary clearance to removes the bacteria. • Quantity or consistency of the sinonasal secretions can affect mucociliary clearance and promote bacterial growth. • Once the ostium becomes occluded, a local hypoxia develops in the sinus cavity, and sinus secretions accumulate. This combination of low-oxygen tension and a rich culture medium of secretions allows bacterial growth to occur within the sinus.

  6. Causative micro-organisms in Acute sinusitis: • In adults, The most common pathogens are Streptococcus pneumoniae and Haemophilus influenzae. • In children, similar organisms are seen, with the addition of Moraxella catarrhalis.

  7. DIAGNOSIS:itis primarily clinical: • Signs & symptoms: • 7 Major factors • Facial pain & tenderness. • Headache severe and increase with leaning forward. • Facial fullness. • Nasal obstruction/blockage • Nasal & postnasal discharge (purulent, discolored). • Hyposmia/anosmia • Fever .

  8. RADIOLOGY: CT: • The imaging study of choice today is CT • It is sensitive in demonstrating mucosal thickening and revealing trapped secretions within the sinus cavities. • In orbital complications, CT is generally the better study, unless intracranial complications are suspected as well.

  9. Viral respiratory tract infections and allergy will both cause mucosal thickening in the absence of infectious or chronic sinusitis. • About 40% of normal people without sinonasal complaints will have abnormalities of the sinus mucosa on CT scan that may be transient and not indicative of true disease.

  10. MRI:Indications: • If cranial or intracranial complications is suspected. It clearly demonstrate dural inflammation that would not be appreciable by CT.

  11. COMPLICATIONS OF SINUSITIS: • In the antibiotic era, such complications have become less common, but they still have the potential for serious morbidity or even mortality. • Improved diagnostic modalities and advances in medical and surgical techniques have significantly reduced the risk of blindness or life-threatening intracranial infections.

  12. I. Orbital complications: • Most orbital complications occur in young children, but those in older children and adults are typically more severe and necessitate surgery. • Ethmoiditis most commonly leads to orbital involvement.

  13. 5 Stages: • 1ST stage, pre-septal peri-orbital cellulites: • Consists of eyelid swelling anterior to the orbital septum (septum is a fibrous membrane dividing the eyelid into anterior and posterior chambers) without involvement of the orbital contents. • 2ND stage, orbital cellulites: • Orbital soft tissue becomes involved, a diffuse process of inflammation without abscess formation. • Patients with this complication are generally proptotic, with some degree of ophthalmoplegia and chemosis.

  14. 3RD stage, sub-periosteal abscess: • Pus accumulates between bone and orbital periosteum. • This will displace the orbit inferolaterally and may cause some proptosis. • Unrecognized or untreated, the process can expand to cause extraocular muscle impairment, chemosis, and loss of visual acuity. • 4TH stage, orbital abscess: • Pus within the orbital tissue. • marked proptosis, limitation of extraocular movement, and visual loss are commonly observed.

  15. 5TH stage, cavernous sinus thrombosis (CST): • Result from extension of ethmoid or sphenoid sinusitis directly or via thrombophlebitis of the ophthalmic vein. • Proptosis, chemosis, ophthalmoplegia, and decreasing visual acuity are the rule. • Process can extend to the opposite side, and bilateral findings are considered a diagnostic hallmark.

  16. II. Intracranial complications: • Most intracranial infections arise from the frontal sinus. • The types of complications that may develop include osteomyelitis of the frontal bone, meningitis, epidural abscess, subdural empyema, and intracerebral abscess

  17. TREATMENT OF SINUSITIS • Antibiotic better amoxil clavunic acid for two weeks. • Decongestant & mucolytic. • Analgesia Last one

  18. Thanks for your attentionwww.zahrawi.ly الحمد لله

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