1 / 24

Rheumatic heart disease

Rheumatic heart disease. Mitral stenosis. Valvular heart disease. Rheumatic Age related congenital. Mitral valve. Stenosis Regurgitation Prolapse. Mitral stenosis. 2/3 females Usually rheumatic Rarely congenital 40% of all RHD . Structural defects.

maree
Télécharger la présentation

Rheumatic heart disease

An Image/Link below is provided (as is) to download presentation Download Policy: Content on the Website is provided to you AS IS for your information and personal use and may not be sold / licensed / shared on other websites without getting consent from its author. Content is provided to you AS IS for your information and personal use only. Download presentation by click this link. While downloading, if for some reason you are not able to download a presentation, the publisher may have deleted the file from their server. During download, if you can't get a presentation, the file might be deleted by the publisher.

E N D

Presentation Transcript


  1. Rheumatic heart disease Mitral stenosis

  2. Valvular heart disease • Rheumatic • Age related • congenital

  3. Mitral valve • Stenosis • Regurgitation • Prolapse

  4. Mitral stenosis • 2/3 females • Usually rheumatic • Rarely congenital • 40% of all RHD

  5. Structural defects • Diffusely thickened –fibrous tissue /calcified deposits • Mitral commisures fuse • Corde tendinae fuse /shorten • Narrowing of the apex of funnel shaped valves

  6. Calcification of slender valves immobilises the leaflet and narrows the orifice –thrombus formation –arterial thrombus from calcified Valves

  7. Pathophysiology • Normal mv –dia -4-6 cm2 • <2 cm 2-atrial to ventricular flow is maintained by increased av pressure gradient –the hallmark of ms • <1 cm2 –LAP should be atleast 25mm hg is required to maintain normal output .

  8. Increased Lap --------increased pulm pressure ------increased capillary pressure -----decreased pulm compliance -------exertionaldyspnoea. • Increased heart rate –decreased transvalvular gradient ----increased LAP • Lv diastolic pressure in normal in ms • Co is normal at rest ---at exercise –decreased co.

  9. $ • Clinical /hemodynamic Features –influenced by • Passive backward transmission of LAP • Pulmonary arteriolar constriction • Intertitial edema • Organic obliterative changes in the pul vascular bed • Phtn----Tr------rt sided failures---bornheimeffect

  10. symptoms • Carditis---ms-----2 decades, • Dyspnoea on exertion ----4 th decade—progressive worsening to death---2-5 yrs • Doe ,orthopnoea ,pnd,arrthmia-premature atraial complex,paroxysysmal tachycardia,flutter,fibrilation • Haemoptysis –increased pulm venous pressure

  11. Recurrantpulm embolism • Pulm infection • Endocarditis • Chest pain -10% • Thrombus formation in the left atrium-af—appendages of LA • Pedunculated thrombus –ball valve thrombi • -syncope-angina –changing ascultatory signs

  12. On examination • Malar flush-pinched blue facies • JVP-a wave prominence –af –a wave absent • Palpation-tapping apical impulse ,s1 loud,palpable ,s2 p2 loud • Diastolic thrill • Auscultation-s1 accentuated /snapping –delayed –mv doesn’t close till LVP>LAP • Qs prolongation ,p2 loud

  13. A2-p2-os -0.05-0.12 • P2-os –severity of ms • Intensity of s1/os –pliability of leAFLET • MDM after os • Duration correlates with ms severity • S1-closure of mitral /tricuspid valve

  14. Intensity of s1 • Pos of mv at onset of vent systole • Rate of increase in LAP • Degree of structural damage of the valve • Amt of tissue bet heart and sthetoscope

  15. S1 loud –diastole is shortened by tachycardia • S1 split -10-30 msec • S1 –m1t1-----prolonged in rbbb • t1m1 –severe ms ,left atrial myoma lbbb

  16. Mitrl regurgitation

  17. etiology • Chronic rhd –severe mr- 1/3 • Seen in males mostly • Rheumatic process-rigidity,deformity,retraction of the valve cusps-commisural fusion • Congenital-endocardial cushion defects • Fibrosis of papillary muscles in MI • Ischeamia –paplillary dysfn

  18. Lv dilated in DCM • HOCM-ant displace ment of the ant leaflet • Mitral prolapse –MR • Acute MR-inf endocarditis

  19. pathophysiology • Clinical pic depends on p-v relation ship of LA AND PUL -VENOUS BED • Increased LAP-Increased pulm edema • Effective forward pressure of lv decreases • Inc-LA volume –due to atrial compliance • Low cardiac out put • Atrial fibrillation

  20. SYMPTOMS • FATIGUE • Doe • Orthopnea • Pnd • Haemoptysis • Sys embolism • Rh f-jvp inc,tr,phtn,hep congestion

  21. Physical examination • Sys thrill-left apex • Hyperdynmic apical impulse • Laterally displaced • Palpable p2 • Parasternal heave

  22. auscultation • S1-absent/softor buried in systolic murmur • Decreased co-aorta closes early-a2 early-wide spliting of s2 • Os –indicates ms • Gallop rhythm • Pansystolic murmur

  23. lab • Ecg –sinus rhythm ,prominent p waves ,af lvh • Echo • Cxr-kerley b lines

  24. management • Medical • Dec exertion • Dec NA intake • Diuretics • Digitalis/vasodilators-inc co • Ace inhibitors /hydralazine • Surgical-valve replacement

More Related