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Chronic adaptations to training.

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Chronic adaptations to training.

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  1. Chronic adaptations to training.

  2. Chronic adaptations to training. • What is Chronic adaptations to training? • How long does it take? • Read Chapter 11 Nelson Physical Education

  3. Chronic adaptations to training • Athletes train to adapt their bodies to a particular sport/activity. • These changes or adaptations in the body is specific to the training method/s applied • Adaptation = “a long-term physiological change in response to training loads that allows the body to meet new demands. • If training load is not sufficient to challenge the body, no adaptations occur and a plateau will occur.

  4. Aerobic and anaerobic training methods

  5. Typical Metabolic and physiological values for healthy trained and untrained men

  6. Aerobic Energy Systems Adaptations • ↑’ed levels of anaerobic enzymes and fuels including; • ATP • PC • Glycogen • ↑ in glycolytic capacity – “the ability to break down glycogen via key enzymes that facilitate glycolysis” • ↑’ed amounts and activity levels of enzymes involved in anaerobic glycolysis (mainly fast twitch fibres) • ↑’ed ability to produce blood lactate during maximal exercise. Results in an increase in glycogen stores and glycolytic enzymes.

  7. Aerobic Energy Systems Adaptations • ↑ in oxygen uptake, transport and utilisation • ↑’ed enhanced fat breakdown (from an ↑ in fat metabolising and ↑’ed fat mobilising enzymes) • Improved fatty acid oxidation and respiratory ATP production • ↑ reduced CHO use during sub-maximal exercise. Therefore these factors assist in glycogen sparing which leads to better endurance

  8. Aerobic Energy Systems Adaptations Aerobic training also causes important changes in the anaerobic threshold! • Ie the point where LA beings to accumulate. Generally this id at 85% of Max HR. But with the aerobic training comes; • ↑’ed capillarisation • ↑’ed mitochondria density • ↑’ed oxidative enzymes • Structural changes to the cardiovascular system. Therefore the anaerobic threshold can improve closer to 90% of Max HR.

  9. Aerobic Energy Systems Adaptations OBLA – Onset of Blood Lactate concentration shows and an increase equal to 4.0mM

  10. Trained Athlete Question 1 Put an X on the point of Lactic threshold. Question 2 What would the red line (blood lactate) look like for an untrained athlete? Question 3 Place a ▲ on the OBLA for both trained and untrained people

  11. Untrained Athlete Lactic Threshold OBLA

  12. Questions • a. List 3 methods of training that will predominantly bring about training adaptations to the aerobic system b. List 3 methods of training that will predominantly bring about training adaptations to the anaerobic system • Under sub-maximal aerobic conditions explain why it is better to use Fats over CHO’s as a fuel source? • What is the relevance of anaerobic threshold? How does it respond to aerobic training? • Resting heart rate is one of the few variables that decrease as a result of training especially aerobically. It has been said that this is because the heart is more efficient. What does this mean? • What does the vascular system refer to?

  13. Cardiovascular Training adaptations ↑’ed oxygen delivery to working muscles due to; • ↑’ed Plasma • ↑’ed Haemoglobin • ↑’ed Total blood volume • ↑’ed ventricle size • ↑’ed venous return • ↑’ed myocardial contractility • ↑’ed max stroke vol. • ↑’ed max cardiac output • ↑’ed effectiveness of cardiac output • ↑’ed peripheral blood flow • ↑’ed blood flow to working muscles • ↑’ed capillarisation • ↑’ed Arteriovenous oxygen difference (A-VO2 diff)

  14. Cardiovascular Training adaptations – Cardiac Hypertrophy • Greatest improvements are attained in first 3 months. After 3 years of training only very slight improvement

  15. The heart muscle itself P251. text

  16. Increased plasma, haemoglobin and myoglobin volumes • Increased plasma, haemoglobin and myoglobin volumes contribute to improved oxygen transport and temperature regulation during exercise. • Haemoglobin helps transport oxygen throughout blood vessels • Myoglobin assists in moving oxygen from cell membranes to the mitochondria.

  17. Changes in Heart Rate • Resting and sub-maximal HR’s will decrease as a result of aerobic training. • Mainly due to • ↑ in stroke volume • ↑ in Q • Therefore to supply the same amount of oxygen, the heart needs fewer beats per min. Therefore the heart becomes more efficient. It pumping the same amt of blood with fewer beats.

  18. Changes in Heart Rate STROKE VOLUME

  19. Changes in Heart Rate How much blood the heart is pumping out per minute. So…. Which heart before/after training is working harder? ________________________________________ Why? _____________________________________________________ __________________________________________________________

  20. Changes in Heart Rate • Improved heart rate recovery • Trained individuals will return to resting HR’s faster than an untrained individual.

  21. Complete

  22. Without training males; males 20-22 L/min and females 15-16 L/min. With training values have exceeded 30 L/min Increased Cardiac Output at maximum workloads

  23. Blood Pressure Reminder Systolic Blood Pressure Pressure on the arteries following contraction of ventricles as blood is pumped out of the heart Diastolic Blood Pressure Pressure in the arteries when the heart relaxes and ventricles fill with blood

  24. Blood Pressure • The greatest changes occur with the systolic pressure. • This is a direct result of; • Improved capillarisation • Improved elasticity of blood vessels • ↑’ed HDL’s (high-density lipoproteins, breaking down fatty deposits/plaque built on inside of arterial walls) • People with high Blood pressure place a high stress on the cardiovascular system.

  25. Oxygen extraction: a-V02 difference • a-V02 difference = Arteriovenous oxygen difference: “difference in oxygen consumption when comparing that in the arterioles to the venules, and an indirect measure of how much oxygen muscles are using” • An ↑in a-V02 difference results in • More blood being pumped to active muscles (especially slow-twitch) • Muscle fibres better at extracting and processing oxygen as a result of ↑’ed mitochondria numbers, more oxidative enzymes and ↑’ed levels of myoglobin. • All of this is due to the oxygen demands of the muscles

  26. a-V02 difference 12 mL/100mL

  27. a-V02 difference 18 mL/100mL

  28. Respiratory Adaptations • Tidal volume – amount of air inspired and expired during normal breathing. Number per minute decreases at rest • Minute Ventilation – at rest MV decreases, at Maximum 02 uptake MV increases to allow more air into the lunge and greater breathing frequency. • Improved lung function – increased surface area for the gas exchange

  29. Respiratory Adaptations • Aerobic capacity – “the maximum amount of oxygen the body can take in, transport and use” • can increase from 10-25% in the first 6 months with intense aerobic training. • Over 2 years can increase 40%.

  30. Respiratory Adaptations Page 255 of text

  31. Questions • Aerobic training improves VO2 max of athletes as an adaptation to the training. List at least two changes that result in this improvement. • Activity 2 - page 255 of text • Review questions 7-10

  32. Aerobic level Anaerobic level Muscular Training Adaptations Vs

  33. Genetics a big advantage to start with x amount of fibre percentage You are born with x amount of fast and slow twitch fibres. BUT you can train and gain more of one type. MYTH – “with training you can change from fast twitch to slow twitch or vice versa.” IMPOSSIBLE HOWEVER – fast twitch fibres have been known to take on slow twitch characteristics in response to aerobic training Muscular Training Adaptations

  34. Muscular Training Adaptations - Anaerobic level • Strength/power and speed training • Greatest adaptations occur at tissue level • Muscular Hypertrophy • Fast twitch fibres (type II) • High intensity loads, low reps • Males have greater results due to presence of testosterone

  35. Muscular Training Adaptations - Anaerobic level • Increased energy substrate levels in muscle • ATP • CP • Glycogen • Increased ATP-PC splitting and resynthesis • Mainly due to sprint training • Increase in the level of enzymes responsible for this Substances that are the most readily available fuel source of muscular energy

  36. Muscular Training Adaptations - Anaerobic level • Increased Glycolytic capacity • Enzymes responsible for the breakdown of glycogen show increased concentration • Sprint training • Increased ventricle thickness • - Don’t forget the heart is a muscle as well!

  37. Muscular Training Adaptations - Anaerobic level • Increased contractile proteins in muscles • Strength training tends to add to the protein of the muscle that generates tension hence greater force generated at any given speed • Increased myosin ATPase • This is the enzyme that splits ATP to yield energy for muscular contractions • More of this enzyme = more energy released allowing contractions to occur at a quicker rate

  38. Muscular Training Adaptations - Anaerobic level NEW THANKS TO THE RESEARCH SCIENTISTS • Increased muscle buffering • Greater LA tolerance is evident when vigorous anaerobic training has taken place possibly due to body’s improved capacity for acid-base regulation • Muscle hyperplasia (new fibres formed) • We knew that muscle fibres increased in size but anaerobically they have been seen to also increase in number via longitudinal splitting • Changes occur due to chronic overload to the skeletal muscle

  39. Muscular Training Adaptations - Aerobic level • Increased Mitochondria density and number • Aerobic powerhouse of the body • Where ATP production occurs • Results in an increase in the capacity for aerobic metabolism from oxidation of FFA’s and CHO for endurance work • Mitochondria numbers can double under the right training • NOTE: anaerobic resistance training will reduce this effect • Eg – Soccer players Vs Body builders

  40. Muscular Training Adaptations - Aerobic level • Increased myoglobin stores • Similar to haemoglobin (transports oxygen in the blood) Myoglobin provides intramuscular oxygen storage. • More myoglobin = more oxygen can be stored at the muscles • Hence more ATP production.

  41. Muscular Training Adaptations - Aerobic level • Improved oxidative capacity Via increased oxidative enzymes (kreb’s cycle) • Faster ATP production • Improved capillary density • Greater oxygen exchange due to greater surface area available. • Improvements in VO2 max

  42. Muscular Training Adaptations - Aerobic level • Increased use of Fats during sub-maximal exercise • To inhibit CHO use • Mainly during the first 30 minutes of exercise • More CHO hence glycogen available for later during endurance performance • Glycogen sparing

  43. Muscular Training Adaptations - Aerobic level • Increased stores and use of intramuscular triglycerides • (Triglycerides, which are chains of high-energy fatty acids, provide much of the energy needed for cells to function.) • Assists with glycogen sparing • Ideal fuel for low-intensity and sub-maximal exercise

  44. Muscular Training Adaptations - Aerobic level • Increased muscle glycogen synthase and storage • Glycogen synthase is the enzyme responsible for converting glucose to glycogen. • Aerobic glycolysis is faster and more efficient • Therefore increase in performance

  45. Adaptations are reversible • When an athlete ceases training they experience a rapid loss of their acquired adaptations. • And endurance athlete confined to bed for 3 weeks can lose • Max stroke volume • Q • Aerobic capacity 1% per day

  46. Questions • What is the main difference between fast and slow twitch fibres? • Muscle hypertrophy occurs in response to both aerobic and anaerobic training. Briefly discuss what this means and how it brings about improved performance levels under each situation. • How do mitochondria bring about improved aerobic performances? • As a result of aerobic training, muscles ‘learn’ to make earlier and greater use of fats as a fuel (especially under sub-maximal exercise conditions). Explain how this leads to improved endurance performances.