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Effects of MEK Inhibition on ERα66 and GAPDH in MCF7 Cells

This study investigates the impact of MEK inhibition at 1μM on ERα66 expression and GAPDH levels in MCF7 breast cancer cells. The insights gained from this research could contribute to understanding the therapeutic potential of MEK inhibitors in hormone-responsive cancers. By analyzing protein levels and signaling pathways, we aim to clarify the role of ERα66 in the context of MEK inhibition and its implications for cancer treatment strategies. This work highlights the intricate relationship between growth factor signaling and estrogen receptor modulation.

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Effects of MEK Inhibition on ERα66 and GAPDH in MCF7 Cells

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  1. ISHIKAWA MCF7 - + MEKi 1μM ERα66 GAPDH

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