'Abdominal aortic aneurysm aaa' diaporamas de présentation

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Vascular Problems, Stroke, Aneurysms, and HTN Crisis

Vascular Problems, Stroke, Aneurysms, and HTN Crisis. By Diana Blum MSN MCC NURS 2140. Vascular Disorders. Common disorders in America: hypertension atherosclerosis arterial occlusive disease abdominal aortic aneurysms (AAA) deep vein thrombosis (DVT)

832 views • 83 slides

An-Najah National University Faculty of Nursing Dr Aidah Abu Alsoud Alkaissi RN, BSc, MSc, PhD

Nursing Care Plan of Aortic Aneurysm. An-Najah National University Faculty of Nursing Dr Aidah Abu Alsoud Alkaissi RN, BSc, MSc, PhD. What is Aortic Aneurysm ?. Is a localized sac or dilation at a weak point of the aorta to a size greater than 1.5 times its normal diameter.

809 views • 68 slides

Age Appropriate Screening

Age Appropriate Screening. Stephen J. Titus MD. Objectives. Review the US Preventive Services Task Force recommendations for Cervical Cancer Breast Cancer Colon Cancer Prostate Cancer Abdominal Aortic Aneurysm Osteoporosis screening Testable Tid-Bits Practice Questions. USPSTF.

1.65k views • 56 slides

EndoVascular Aortic Aneurysm Repair (EVAR)

EndoVascular Aortic Aneurysm Repair (EVAR). Dr. Thomas Lam PYNEH. Definition of an Aneurysm. Focal arterial dilatation based upon absolute measurements of diameter A localized dilatation of an artery with an increase in diameter of 50% or more than that of the non-dilated adjacent vessel

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579 views • 28 slides

Advanced Journal of Vascular Medicine

The aim of this review was to better defi ne pathogenesis of Abdominal Aortic Aneurysm (AAA) in patients with increased homocysteine plasma levels. AAA is a non-frequent clinical condition that could evolve towards death for its rupture. It has traditionally been regarded as a consequence of atherosclerosis. That is induced by several risk factors, such as age, family history, smoking, hypertension, diabetes, dyslipidemia and others. Among these risk-factors, elevated plasma levels of homocysteine is also found. Hyperhomocysteinemia (HHcy) can cause breakdown in the elastic fi bers of the aortic wall, through degradation of Extracellular Matrix (ECM). That causes the activation of Matrix Metalloproteinases (MMPs) contrarily acting to their Tissue Inhibitors (TIMPs). The imbalance of TIMPs/MMPs ratio with the MMPs prevalence, in turn, induces a proteolytic effect (elastolysis) of the aortic wall and AAA formation. Subsequently, the activation of coagulative process may cause intraluminal thrombus formation that can accelerate or delay the aneurysmatic rupture. But, numerous doubts exist around the role of HHcy in the dilatation of abdominal aortic tract, the importance of intraluminal thrombus for AAA rupture or protection, and the slowing down effect of TIMPs in the AAA formation. Thus, other researches are needed to defi nitively solve these uncertainties.

51 views • 5 slides

Advanced Journal of Vascular Medicine

The aim of this review was to better defi ne pathogenesis of Abdominal Aortic Aneurysm (AAA) in patients with increased homocysteine plasma levels. AAA is a non-frequent clinical condition that could evolve towards death for its rupture. It has traditionally been regarded as a consequence of atherosclerosis. That is induced by several risk factors, such as age, family history, smoking, hypertension, diabetes, dyslipidemia and others. Among these risk-factors, elevated plasma levels of homocysteine is also found. Hyperhomocysteinemia (HHcy) can cause breakdown in the elastic fi bers of the aortic wall, through degradation of Extracellular Matrix (ECM). That causes the activation of Matrix Metalloproteinases (MMPs) contrarily acting to their Tissue Inhibitors (TIMPs). The imbalance of TIMPs/MMPs ratio with the MMPs prevalence, in turn, induces a proteolytic effect (elastolysis) of the aortic wall and AAA formation. Subsequently, the activation of coagulative process may cause intraluminal thrombus formation that can accelerate or delay the aneurysmatic rupture. But, numerous doubts exist around the role of HHcy in the dilatation of abdominal aortic tract, the importance of intraluminal thrombus for AAA rupture or protection, and the slowing down effect of TIMPs in the AAA formation. Thus, other researches are needed to defi nitively solve these uncertainties.

54 views • 5 slides