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This guide focuses on pediatric emergencies, highlighting the unique developmental variances in children that affect emergency treatment. Key insights include the management of hypovolemic and distributive shock, the importance of triage in emergency departments, and the physiological differences that necessitate specific interventions. Highlights include signs of shock, assessment techniques, and appropriate fluid resuscitation protocols. Understanding these differences can significantly improve patient outcomes and ensure timely and effective care for young patients in critical situations.
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Pediatric Emergencies Jan Bazner-Chandler RN, MSN, CNS, CPNP
Developmental and Biologic Variances • Cricoid is the narrowest portion of the airway: no cuffed ET tubes in children under 8 years of age ET cuffed
Developmental and Biologic Variances • Total blood volume is smaller – small blood loss may led to hypovolemia and impaired profusion • Healthy children in shock will maintain blood pressure until more than 25% of blood volume is lost • Tachycardiaand delayed capillary refill are early signs of shock • Decreased blood pressure is late sign
Developmental and Biologic Differences • Respiratory arrest is more common in pediatric population • Respiratory rate below 10 or above 60 are sign that child may be headed for respiratory arrest without interventions
Triage • To “pick or sort”. • Goals of triage: • Rapidly identify seriously injured. • Prioritize all patients using the emergency department. • Initiate therapeutic measures.
Triage Classification • Resuscitation • Emergent- needs to be seen within 10 minutes • Urgent – need to be seen within 30 to 60 minutes • Semi-urgent – need to be seen within 1to 2 hours • Non-urgent – need to be seen within 2 to 3 hours
Assessment • Across-the-room assessment • Chief complaint • Brief history (AMPLE Mnemonic) • Allergies • Medications • Past medical history • Last meal • Events surrounding the incident
Focused Physical Assessment • Airway • Breathing • Circulation • Disability • Exposure • Full vital signs • Family presence • Give comfort • Head-to-toe assessment • Inspect • Isolate
Test and Procedures • CBC with differential: infection and lack of immune response • Type and cross match: blood type • Serum electrolytes: electrolyte imbalance • Radiographs: chest, abdomen, bones • Computed tomography – CT scan: detects bleeding or masses
Shock • Hypovolemic shock • Distributive • Cardiogenic • Obstructive Note: cardiogenic and obstructive more common in the adult
Shock • The earlier you can recognize shock, establish priorities, and start therapy, the better the child’s chance for a good outcome.
Hypovolemic Shock • Most common cause of shock in children • Fluid and electrolyte losses associated with fluid loss • Blood loss from trauma • Etiology: caused by inadequate volume relative to the vascular space
Hypovolemic Shock • Most common cause of shock in children worldwide • Fluid loss due to diarrhea is the leading cause • Other causes • Hemorrhage • Vomiting • Inadequate fluid intake • Osmotic diuresis (eg diabetic ketoacidosis • Third space losses (fluid leak into tissues • Burns • Sepsis
Physiology of Hypovolemic Shock • Characterized by decreased preload leading to reduced stroke volume and low cardiac output. • Compensatory mechanisms are tachycardia, increased contractility, and increased systemic vascular resistance.
Hypovolemic shock: Assessment • Cardiovascular • Tachycardia • Normal blood pressure or hypotension with a narrow pulse pressure • Prolonged capillary refill > than or equal to 2 seconds • Weak, thready or absent peripheral pulses • End-organ function • Cool to cold, pale diaphoretic skin • Changes in mental status • Oliguria
Interdisciplinary Interventions • IV fluids 20 mL/kg bolus of Crystalloid Solution • 0.9% normal saline • Ringer’s lactate • If signs of inadequate profusion after 2 or 3 boluses administer 10 mL / pg packed red blood cells • Control bleeding
Distributive Shock • Septic shock • Anaphylactic • Neurogenic shock (head injury, spinal injury)
Septic Shock • Most common form of distributive shock. • Caused by infectious organisms or their byproducts that stimulates the immune system and trigger release or activation of inflammatory mediators. • Uncontrolled activation of the inflammatory mediators can lead to organ failure, particularly cardiovascular and respiratory failure, systemic thrombosis and adrenal dysfunction.
Assessment Findings • History or infection • History of poor feeding • Physical findings • Tachycardia: HR > 2 standard deviations above normal for age • Fever: > 38.5 or < 36 (neonate may be hypothermic) • Tachypnea: RR > 2 standard deviations above normal for age • Altered mental status - lethargy • Petechiae / or purpura • Poor peripheral perfusion (capillary refill less than 2 seconds) • Hypotension – late sign
Laboratory Values • WBC • Greater than 12,000 • Lower than 4,000 or more than 10% immature neutrophils • Platelets in the acute phase may be elevated due to inflammation. • Platelets may decrease in the case of DIC
Interdisciplinary Interventions • Isolate if indicated • IV fluids (crystalloid solution) to restore circulating volume • Inotropic agents as needed • Norepinephrine – alpha receptor agonist causes peripheral arterial vasoconstriction • Dopamine – beta receptor agonist to increase cardiac output • Cultures: blood, spinal fluid, urine • Broad spectrum antibiotics: MRSA • If hypoglycemic – IV glucose
Sepsis with ARDS • Acute respiratory distress syndrome • Mechanical ventilation • Aggressive antibiotics to treat bacterial infection • Methylprednisone – anti-inflammatory
Anaphylactic Shock • Results from a severe reaction to a drug, vaccine, food toxin, plant, venom or other antigen. • It is characterized by venodilation, systemic vasodilation, and increased capillary permeability combined with pulmonary vasoconstriction. • Vasoconstriction increased right heart work and may add to hypotension by reducing the delivery of blood from the right ventricle to the left ventricle
Assessment Findings • Anxiety or agitation • Nausea and vomiting • Urticaria (hives) • Angioedema (swelling of face, lips and tongue) • Respiratory distress with stridor or wheezing • Hypotension • Tachycardia • What is first drug of choice?
Poisoning • The fifth leading cause of death in children younger than 5 years • Overdose in infants are often the result of therapeutic overdosing • Children younger than 6 years • Cleaning substances, analgesics, topical agents, cough and cold preparations • Adolescents drug experimentation and suicide attempts Questions: Why is OD on Tylenol (acetaminophen) a problem?
Poisoning • Over a million children are poisoned annually. • Ages of risk are 2 to 4 years and adolescents. • Common poisons ingested: • Iron, lead, acetaminophen, hydrocarbons, liquid Drano, and plants.
Assessment • #1 Look at the child • May present with no symptoms to coma
Focus History • What was ingested? • How much was ingested? • When did it occur? • What therapy was initiated before arrival in the ED?
AAP Recommendations • AAP – American Academy of Pediatrics • Syrup of Ipecac no longer be used routinely in the home to induce vomiting. • Research has failed to show benefit for children who were treated with Ipecac. • Prevention is the best defense against unintentional poisoning
Parent Teaching • Post the universal phone number for poison control center near the telephone • 1-800-222-1222 • Call 911 in the case of convulsions, cessation of breathing or unconsciousness • Do not make your child vomit
Emergency Treatment • Always assess the child to determine the care: airway, breathing, LOC • History of what substance was swallowed • Ask parent to bring in container or sample of substance swallowed • Activated charcoal may be given to help absorb substance ingested
Lead Poisoning • There are about 1.7 million children with elevated lead levels. • A large proportion are poor, African-American, Mexican-American, and living in urban areas. • Children are more susceptible because they absorb and retain lead.
Lead Poisoning • Lead interferes with normal cell function, and adversely affects the metabolism of vitamin D and calcium. • Clinical manifestations depend on degree of toxicity. • Neurologic effects include decreased IQ scores, cognitive deficits, impaired hearing, and growth delays.
Lead Poisoning • Sources of lead: • Lead based paint • Soil and dust • Drinking water from lead lined pipes • Food growth in contaminated fields • Contamination from occupations or hobbies
Lead Levels • Blood lead levels between 10 and 19 ug/dL are typically asymptomatic • Teaching about hazards of lead • Blood levels between 20 to 44 ug/dL may present with increase motor impairment and lethargy (poor school performance) • Home assessment • Chelation therapy may be indicated • Levels greater than 70 ug/dL are considered an emergency
Prevention of Lead Poisoning • Washing hands and toys • Low-fat diet • Check home for lead hazards • Regularly clean home • Take precautions when remodeling or working on old cars, furniture, or pottery. • Call 1-800-424-lead for guidelines