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ACS and the 12 Lead ECG

ACS and the 12 Lead ECG. The 12 Lead ECG is at the center of the decision pathway in the management of patients with ischemic chest pain. AHA Guidelines 2000. Scenario #1.

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ACS and the 12 Lead ECG

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  1. ACS and the 12 Lead ECG

  2. The 12 Lead ECG is at the center of the decision pathway in the management of patients with ischemic chest pain. AHA Guidelines 2000

  3. Scenario #1 • 55 y/o male c/o sub-sternal chest pressure that started while exerting himself. Pain radiates to his neck/jaw. Pain unrelieved with rest or patients own NTG. • Patient is cool, and pale. • DX?

  4. Scenario #2 • 60 y/o female c/o dull, aching, pain in her chest that radiates to her left breast. Sts pain has been constant for past hour, getting increasingly worse. Hx of HTN, NIDDM, Angina. Sts pain is like previous angina attack, just worse. • Patient is cool, pale, diaphoretic. • DX?

  5. What similarities do these two scenarios share? • How can their treatment both be different. • How will the 12 lead guide our treatment of these patients?

  6. Introduction • Acute Coronary Syndrome is a group of disease process’s that signs and symptoms mimic each other. • Although similar, they are not the same. • The treatment for ACS is based on the patient’s current “stage” in the disease process

  7. A C S S T A G E S

  8. Stable Angina • A flow/demand imbalance between reduced blood flow through a narrowed artery, and increased demand.

  9. Unstable Angina/Non-Q Wave Infarction • Symptoms of angina that are new or increasing or that occur at rest. Seldom relieved with rest, O2, or NTG. • Symptoms are usually due to platelet aggregation in narrowed arteries with chronic atherosclerotic occlusions.

  10. Unstable Angina/Non-Q Wave Infarction Unstable Angina can be complicated by the release of micro-emboli that occlude distal micro-vasculature.

  11. Q-Wave Infarction • Complete formation of thrombus in an artery. • Spontaneous lysis of the clot can occur, but often too late to salvage the heart muscle.

  12. Indicative Changes • Ischemia: Symmetrically inverted T waves or down sloping ST segment depression greater then 1mm • Injury: ST segment elevation greater then 1mm • Infarct: Pathological Q wave formation

  13. Ischemia

  14. Injury/Infarction

  15. Indicative Changes • Based on the patients 12 lead ECG, they will be placed into 1 of 3 treatment categories that are aimed at correcting the specific Acute Coronary Syndrome present.

  16. Treatment Categories

  17. Why Do We Care? • The only ACS disease process that benefits from Thrombolytic therapy are those that are caused by complete occlusion of an artery. • Recognition of ST SegmentElevation ACS is the first step in a race against the clock.

  18. Treatment Strategies

  19. Treatment Strategies

  20. Treatment Strategies

  21. Treatment Strategies NO YES

  22. Scenario Review

  23. Scenario #1 • 55 y/o male c/o sub-sternal chest pressure that started while exerting himself. Pain radiates to his neck/jaw. Pain unrelieved with rest or patients own NTG. • Patient is cool, and pale.

  24. Scenario #1 • Does this patient meet ECG requirements for thrombolysis? • What stage of ACS can you predict from the presenting 12 lead? • What complications can you expect to see if the patient’s condition worsens?

  25. Scenario #2 • 60 y/o female c/o dull, aching, pain in her chest that radiates to her left breast. Sts pain has been constant for past hour, getting increasingly worse. Hx of HTN, NIDDM, Angina. Sts pain is like previous angina attack, just worse. • Patient is cool, pale, diaphoretic.

  26. Scenario #2 • Does this patient meet ECG requirements for thrombolysis? • What significance do the tall T waves suggest? • What complications can you expect to see if the patient’s condition worsens?

  27. Exceptions to the Rules • Some subsets of patients may be eligible for Fibrinolytic therapy even though they do not present with ST segment elevation. • Posterior current of injury • Tall, hyperacute t waves

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