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Placental Abruption

Placental Abruption. Pathophysiology. Small vessel rapture Bleeding lifts placenta because uterus is unable to contract and control bleeding. Further Haemorrhage dissects off chorio decidual space Due to uterine contractions blood flows through membranes and uterine walls separating them.

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Placental Abruption

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  1. Placental Abruption

  2. Pathophysiology • Small vessel rapture • Bleeding lifts placenta because uterus is unable to contract and control bleeding. • Further Haemorrhage dissects off chorio decidual space • Due to uterine contractions blood flows through membranes and uterine walls separating them. • Blood escape through vagina – revealed haemorrhage • Tissue Damage and blood clots thrombo plastin – escape into maternal circulation through open sinuses – activate coagulation mechanism – fibrinogen converted to fibrin. –

  3. other factors are also consumed causing consumption coaglopathy.- there is deficiency of factor 5,8 and 13 and plateless. Intra vascular coagulation fibrinoliases – FDP has anti thrombo effect and inhabit platelet aggregation. • Renal perfuson is decreased .actuate oliguria and proteinuria. So pathological features are hypofibrenogina and bilateral renal cortical and renal failure.

  4. Clinical Features • External Bleeding, pain, hard tender uterus. • Fetus is usually dead. • Hypovolemic Shock.

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