'Beta oxidation' diaporamas de présentation

LIPID METABOLISM

LIPID METABOLISM. December 2007. Lipid Digestion. Monogastric Digestion. Challenges 1.Lipids are not water soluble 2.Triglycerides too large to be absorbed Digestive solution 1.Triglycerides mix with bile and pancreatic secretions 2.Emulsification and digestion. Bile.

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Water-Soluble Vitamins

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Oxidation of fatty acids and K etogenesis

Oxidation of fatty acids and K etogenesis. Dr.S.Chakravarty M.D. Specific Learning Objectives . At the end of this lecture students will be able to :-

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Oxidation of Fatty Acids

Oxidation of Fatty Acids. Fatty acids are an important source of energy and adenosine triphosphate (ATP) for many cellular organisms. Excess fatty acids, glucose , and other nutrients can be stored efficiently as fat .

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Review #2 - Cellular Structure

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Overview of intracellular metabolism What are the 4 Stages of aerobic energy production?

Mitochondria and the TriCarboxylic Acid Cycle (TCA or Citric acid cycle) are central to energy production in eukaryotic cells! 10/8 and 10/13. Overview of intracellular metabolism What are the 4 Stages of aerobic energy production? How are mitochondrial organized inside?

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This Content is for Test #3, not Test #2 Friday 10/7 Overview of intracellular metabolism

Mitochondria and the TriCarboxylic Acid Cycle (TCA or Citric acid cycle) are central to energy production in eukaryotic cells! 10/3 and 10/5. This Content is for Test #3, not Test #2 Friday 10/7 Overview of intracellular metabolism What are the 4 Stages of aerobic energy production?

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Overview of intracellular metabolism What are the 4 Stages of aerobic energy production?

Mitochondria and the TriCarboxylic Acid Cycle (TCA or Citric acid cycle) are central to energy production in eukaryotic cells! 10/15 and 17. Overview of intracellular metabolism What are the 4 Stages of aerobic energy production? How are mitochondrial organized inside?

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Fatty acids, eicosanoids

Fatty acids, eicosanoids. Fatty acids are straight chain monocarboxylic acids, having short, medium or long chain: C1-26 in nature can be: saturated or unsaturated, cis or trans Source saturated and trans unsaturated FA we synthesize: palmitic acid and elongate it

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BCHM2072/2972

BCHM2072/2972. 2004 THEORY PAPER MCQs 41 - 54. 41. Graph. The graph below does NOT represent: A. The rate of glucose output from the liver B. The liver glycogen content C. The activity of muscle pyruvate dehydrogenase D. The blood insulin level

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Welcome to class of Lipid metabolism Dr. Meera Kaur

Welcome to class of Lipid metabolism Dr. Meera Kaur. Learning objectives. To understand How fatty acids are activated and transported into mitochondria for oxidation. Oxidation of fatty acids. Role of lipoproteins in cholesterol metabolism. Introduction.

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Welcome to class of Lipid metabolism Dr. Meera Kaur

Welcome to class of Lipid metabolism Dr. Meera Kaur. Learning objectives. To understand How fatty acids are activated and transported into mitochondria for oxidation. Oxidation of fatty acids. Role of lipoproteins in cholesterol metabolism. Introduction.

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b Oxidation

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Beta oxidation

Beta oxidation. Fat. Fat taken up from the blood is either stored in the intramuscular triglyceride or used immediately in mitochondria. To be able to use fat immediately in the mitochondria it needs to be broken down using beta oxidation to get in the TCA cycle. Fig 4.1 (p.60).

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ALTERNATE PATHWAYS, CONTROL OF CELLULAR RESPIRATION AND BMR

ALTERNATE PATHWAYS, CONTROL OF CELLULAR RESPIRATION AND BMR. Alternate Pathways. What are the other fuel sources that can be used in Cellular Metabolism?. Fats. Carbohydrates. Proteins. Amino acids. Fatty acids. Glucose. Glycero l. Glycolysis. Glucose. Glyceraldehyde-3- P. NH 3.

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Beta oxidation

Beta oxidation. Fat. Fat taken up from the blood is either stored in the intramuscular triglyceride or used immediately in mitochondria. To be able to use fat immediately in the mitochondria it needs to be broken down using beta oxidation to get in the TCA cycle. Fig 4.1 (p.60).

230 views • 9 slides

METABOLISM OF 8-ISO-PGF2 ? IS IMPAIRED BY CLA IN FIBROBLASTS FROM PATIENTS AFFECTED BY X-ALD

a. IP. b. DIN. b. a. a. a, b. pmoles/million cells. c. b. CLA + IP. CLA + IP. IP. IP. a. control. X-ALD. a. b. b. CD 16:2 from 10,12. a. a. b. b. CD 16:2 from 9,11. nmoles/10 million cells. CLA. CLA. CLA+ IP. CLA + IP. control. X-ALD.

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