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SURVIVAL STUDIES. Mortality rate ? 10%Mortality ? ISSORIF of unstable pelvic fractures ? mortality rate. NATURAL HISTORY 1. Stable fractures
 
                
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1. PELVIC TRAUMA 
2. SURVIVAL STUDIES Mortality rate ? 10%
Mortality ? ISS
ORIF of unstable pelvic fractures ? mortality rate 
3. NATURAL HISTORY 1 Stable fractures 
few major long-term problems
usually mild or moderate pain 
4. NATURAL HISTORY 2 Unstable fractures 
Persistent pain  60%
Nonunion  3.5%
Malunion  4%
Permanent nerve injury  5.5%
Permanent urethral injury  2.5% 
5. Prognostic Factors Degree of initial force
Type of injury (stable or unstable)
Treatment modalities
Associated injuries 
6. Anatomic considerations Bony ring structure  pelvic ring
Ligaments structures  static & dynamic stabilizers 
7. Biomechanical considerations Pelvic stability
Forces acting on the pelvis
Forces transmission to the viscera, vessels, and nerves 
12. Management Polytraumatized patient
Rapid general assessment and resuscitation (esp. type C) 
13. Resuscitation  Massive fluid replacement
Hemorrhage control 
14. Hemorrhage control Pneumatic antishock garment (PASG)
Stabilization of the unstable pelvic disruption
Embolization of the pelvic vessels
Open surgery 
15. Hemorrhage control Early, provisional stabilization of the unstable pelvic fracture
Use safe, simple and quick methods 
16. Indications for Open Surgery Open (compound) fracture.
Major vessel injury.
Patient in extremis from hypovolemic shock.
BP < 60 mmHg secondary to hypovolemic shock, with little or no response to fluid replacement. 
17. Clinical Assessment History
Patient profile
Injury profile
Physical Examination
Look
Wounds, contussions, bleeding genitalia, displacement of pelvis or lower extremities.
Feel and Move
Palpation, traction, rectal and vaginal examination, neurological examination. 
19. Radiographic Assessment Plain Radiography
A-P, inlet, outlet, oblique views.
Tomography 
Nuclear Scanning
CT 
23. Signs of Instability- clinical factors Severe displacement, including rotation of the pelvis and/or shortening of the extremity.
Marked posterior disruption characterized by bruising and swelling.
Gross instability of the hemipelvis on manual palpation.
Associated injuries to viscera, blood vessels, or nerves.
Associated open wound. 
24. Signs of Instability- radiographic factors Displacement of the posterior SI complex >1 cm, either by a fracture, a dislocation, or a combination of both.
The presence of a gap rather than impaction posteriorly.
The presence of avulsion fractures of the sacral or ischial end of the sacrospinous ligament.
Avulsion fractures of the transverse process of the L5, if associated with a posterior gap.
*  Indicating a posterior lesion 
26. Classification Type A  stable
Type B  vertically stable, rotationally unstable
Type C  unstable (rotationally + vertically) 
37. TYPE B2: LATERAL COMPRESSION INJURIES B2-1   Ipsilateral
Anterior and posterior injury
B2-2   Contralateral
Anterior and posterior injury 
45. Pelvic Ring DisruptionManagement 
46. Management Protocol for Pelvic Ring Disruption 
47. Management Protocol for Pelvic ring Disruption 
48. Management Protocol for Pelvic Ring Disruption 
49. Management Protocol for Pelvic Ring Disruption 
51. Provisional stabilization External frame
Pelvic clamp
Skeletal traction (30-40 lb) 
56. Early Provisional stabilization Type B1 (open book fracture)
Type C
Both result in an increased pelvic volume 
57. Early provisional stabilization Reduce the volume of the pelvis
Restore the tamponade effect of the bony pelvis
Hemorrhage control 
58. Early provisional stabilization Maintaining an upright position for proper ventilation 
59. Early provisional stabilization Biomechanically, not strong enough to allow ambulation
Redisplacement usually occurs 
60. Definitive stabilization Stability of the fracture
Risks and benefits of stabilization 
61. Definitive stabilization Type A
Symptomatic treatment
No need for stabilization 
62. Definitive stabilization- type B1 (open book) 
If symphysis is open < 2.5 cm
=> No specific stabilization
If symphysis is open > 2.5 cm
=> ESF or plate 
63. Anterior internal fixation of disrupted symphysis 
Plating is suggested if :
Laparotomy
No fecal contamination
No need for suprapubic drain 
64. Definitive stabilization- type B2-1 
No specific stabilization 
65. Definitive stabilization- type B2-2 (bucket handle) 
If LLD < 1.5 cm
=> No specific stabilization
If LLD > 1.5 cm
=> ESF
If tilt fracture
=> ORIF 
66. Definitive stabilization- type C 
Simple external frame c traction
Complex external frame c or  traction
ORIF 
67. Definitive stabilization Advantages of ORIF :
Biomechanically, strong enough to allow early ambulation
Reduces the malunion or nonunion rates
 
68. Definitive stabilization Disadvantages of ORIF :
Increased bleeding
Wound problems
Nerve injury 
69. Type C injury Indication for anterior internal fixation
Indication for posterior internal fixation 
70. Indications for anterior internal fixation Fracture types
State of the patient
Laparotomy
No fecal contamination
No need for suprapubic drain 
71. Indications for posterior internal fixation State of the patient
Unstable, unreduced posterior SI complex (esp. an unreduced SI dislocation c a gap of > 1 cm)
Posterior open wound (cf. - contraindicated if  the wound is in the perineum)
Associated acetabular fracture requiring ORIF 
72. Timing of open surgery (ORIF) Wait until general condition is stable, usually between 5th and 7th post-op day 
73. Prophylactic antibiotics Routinely given for a minimum of 48 h
Cefazolin 2g/day, IV
Tobramycin 160mg/day, IV 
74. SURGERY- anterior internal fixation Type B1, a 2- to 4-hole reconstruction plate on the superior surface
Type C, two plates at 90 to  each other, if no posterior fixation is planned 
75. SURGERY- posterior internal fixation Sacral fractures, two transiliac bars
SI dislocations, anterior plating or posterior screw fixation
Iliac fractures, interfragmental screws or plates (3.5-mm reconstruction plates) 
76. Early complications Hypovolemia
Thromboembolism
Fat embolism 
77. Late complications Infection
Multiple organ failure